Post by Admin/ Traveler on Nov 3, 2019 16:19:42 GMT
WARNING!!! This may be VERY disturbing to some, as it's about the brain and spirochetes. So, if you find yourself particularly sensitive today, or any day, please do not read past what I have posted at most.
Should anyone wish to see the entire article, I will be happy to send it through the messages center here on this forum. Just ask!
--------------------------------------------------------------------------------------------------------------------------------------------------------
The Seven Provocative Findings of the Dr. Paul H. Duray Research Fellowship Foundation, and what they mean to Medicine and Patients
by Tom Grier , Microbiologist
July 2016
1) Mother-to-child transmission of Borrelia across the womb
2) Finding Borrelia burgdorferi and B. miyamotoi associated with Amyloid
Plaques in Alzheimer’s disease brains
3) Finding Borrelia in Lewy Body Dementia
4) Nematode worms found in the CSF (spinal fluid) of Multiple Sclerosis patients
5) Nematode worms found in Alzheimer’s brains
6) Borrelia found in five deadly brain tumors (Glioblastoma multiforme)
7) Borrelia Mayonii and Borrelia burgdorferi found in human testicle
Since 1975 when Lyme disease was first introduced to the medical literature, it has been surrounded by controversy and misunderstandings. Much of the problem stemmed from trying to understand this disease entirely through antibody tests (serology) based entirely on just one species – Borrelia burgdorferi.
We now know that there are many species of Lyme disease. Borreliosis is not just one disease, it is part of a family of diseases that can no longer be considered separate or isolated from Lyme disease. The best example of this is Borrelia miyamotoi.
It is found in hard-shelled ticks just like Lyme disease, but it is a Relapsing Fever borrelia.
It took over 10 years for microbiologists to place it in the Relapsing Fever category as opposed to the Lyme-genetic grouping.
Not surprising is the fact that B. miyamotoi is found in the human brain right alongside B. burgdorferi. They may look like two separate diseases on paper, but in the human brain they are pathogens and must be eradicated together.
An even more concerning part of the Lyme disease story is that virtually no funding in any country has been put into Borrelia pathology. The fact is Lyme serology simply cannot detect this family of bacteria with any reliability, mostly because Borrelia, once inside the human brain, is behind the blood-brain barrier, and inside human brain cells. As a result, the human immune system can no longer recognize it.
Add to this the fact there are more than a dozen species of Borrelia that cause Lyme disease, many of which can penetrate any tissue, and add a couple Relapsing Fevers that tag along for the ride, and it becomes clear that the Lyme disease blood tests based on Borrelia burgdorferi detection that have been used for 30 + years have become pretty much useless.
(continued in original document - just ask and I'll send it to you in our message center)
Should anyone wish to see the entire article, I will be happy to send it through the messages center here on this forum. Just ask!
--------------------------------------------------------------------------------------------------------------------------------------------------------
The Seven Provocative Findings of the Dr. Paul H. Duray Research Fellowship Foundation, and what they mean to Medicine and Patients
by Tom Grier , Microbiologist
July 2016
1) Mother-to-child transmission of Borrelia across the womb
2) Finding Borrelia burgdorferi and B. miyamotoi associated with Amyloid
Plaques in Alzheimer’s disease brains
3) Finding Borrelia in Lewy Body Dementia
4) Nematode worms found in the CSF (spinal fluid) of Multiple Sclerosis patients
5) Nematode worms found in Alzheimer’s brains
6) Borrelia found in five deadly brain tumors (Glioblastoma multiforme)
7) Borrelia Mayonii and Borrelia burgdorferi found in human testicle
Since 1975 when Lyme disease was first introduced to the medical literature, it has been surrounded by controversy and misunderstandings. Much of the problem stemmed from trying to understand this disease entirely through antibody tests (serology) based entirely on just one species – Borrelia burgdorferi.
We now know that there are many species of Lyme disease. Borreliosis is not just one disease, it is part of a family of diseases that can no longer be considered separate or isolated from Lyme disease. The best example of this is Borrelia miyamotoi.
It is found in hard-shelled ticks just like Lyme disease, but it is a Relapsing Fever borrelia.
It took over 10 years for microbiologists to place it in the Relapsing Fever category as opposed to the Lyme-genetic grouping.
Not surprising is the fact that B. miyamotoi is found in the human brain right alongside B. burgdorferi. They may look like two separate diseases on paper, but in the human brain they are pathogens and must be eradicated together.
An even more concerning part of the Lyme disease story is that virtually no funding in any country has been put into Borrelia pathology. The fact is Lyme serology simply cannot detect this family of bacteria with any reliability, mostly because Borrelia, once inside the human brain, is behind the blood-brain barrier, and inside human brain cells. As a result, the human immune system can no longer recognize it.
Add to this the fact there are more than a dozen species of Borrelia that cause Lyme disease, many of which can penetrate any tissue, and add a couple Relapsing Fevers that tag along for the ride, and it becomes clear that the Lyme disease blood tests based on Borrelia burgdorferi detection that have been used for 30 + years have become pretty much useless.
(continued in original document - just ask and I'll send it to you in our message center)